A patient presents with possible organophosphate toxicity. What would you expect to find upon assessment?

A patient presents with possible organophosphate toxicity. What would you expect to find upon assessment?

• A. Fever
• B. Diaphoresis
• C. Bradycardia
• D. Hypertension

Answer: (B)

In cases of organophosphate toxicity, one of the hallmark signs to expect upon assessment is diaphoresis, or excessive sweating. Organophosphates are a class of chemicals commonly used as pesticides, and they work by inhibiting the enzyme acetylcholinesterase. This inhibition leads to an accumulation of acetylcholine in the synapses, causing overstimulation of the cholinergic system. As a result, patients may exhibit a range of symptoms consistent with increased cholinergic activity. Diaphoresis is a frequent manifestation due to the autonomic nervous system's response to this overstimulation. Other symptoms can include miosis (constricted pupils), salivation, lacrimation, urination, defecation, gastrointestinal distress, and increased bronchial secretions. The sympathetic nervous system can also be stimulated, contributing to excessive sweating. In contrast, while fever can occur due to other mechanisms or complications, it is not a direct consequence of organophosphate toxicity. Bradycardia is typically observed in cases of severe toxicity due to increased vagal tone from the enhanced cholinergic stimulation; however, it can also alternate with tachycardia in certain situations due to compensatory responses. Hypertension is not a common finding with organoph

In cases of organophosphate toxicity, one of the hallmark signs to expect upon assessment is diaphoresis, or excessive sweating. Organophosphates are a class of chemicals commonly used as pesticides, and they work by inhibiting the enzyme acetylcholinesterase. This inhibition leads to an accumulation of acetylcholine in the synapses, causing overstimulation of the cholinergic system.

As a result, patients may exhibit a range of symptoms consistent with increased cholinergic activity. Diaphoresis is a frequent manifestation due to the autonomic nervous system's response to this overstimulation. Other symptoms can include miosis (constricted pupils), salivation, lacrimation, urination, defecation, gastrointestinal distress, and increased bronchial secretions. The sympathetic nervous system can also be stimulated, contributing to excessive sweating.

In contrast, while fever can occur due to other mechanisms or complications, it is not a direct consequence of organophosphate toxicity. Bradycardia is typically observed in cases of severe toxicity due to increased vagal tone from the enhanced cholinergic stimulation; however, it can also alternate with tachycardia in certain situations due to compensatory responses. Hypertension is not a common finding with organoph